The Current Landscape of Atherosclerosis Part 3
Current evidence supports a different, much more dynamic and discontinuous evolution of atheroma plaques. Experimental and human observations support that recruitment of blood leukocytes mediated
Current evidence supports a different, much more dynamic and discontinuous evolution of atheroma plaques. Experimental and human observations support that recruitment of blood leukocytes mediated
Cellular senescence is one of the 9 ‘‘Hallmarlks of Aging”. Some, but not all, senescent cells may acquire what we call a senescence-associated secretory phenotype
The way our genetic material works is that some genes are transcribed, that is, that their information is read and used to create a protein
NAD or nicotinamide adenine dinucleotide is a derivative of vitamin B3, also known as niacin, niacinamide or nicotinamide (soluble form). NAD is used in over
It is very important to know how efficient our cells are when producing ATP (adenosine triphosphate), a process which depends on how efficient each mitochondria
We know that by manipulating genes, altering the reproductive system, lowering caloric intake, modulating hormone levels, and altering the pathways of IGF-1/insulin, among other things,
It has been shown that, with age, mitochondrial function decreases or worsens in different tissues. This implies an accumulation of morphological alterations at the level
Age is the main risk factor for most of the chronic diseases that afflict us in that last third of our lives. The first step
There are studies underway that focus on anti-ageing drugs -such as metformin and nicotinamide adenine (NAD+)- as well as the molecules that elongate telomeres -such
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